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APOBEC1 complementation factor (A1CF) is dispensable for C-to-U RNA editing in vivo.
Snyder EM, McCarty C, Mehalow A, Svenson KL, Murray SA, Korstanje R, Braun RE. Snyder EM, et al. RNA. 2017 Apr;23(4):457-465. doi: 10.1261/rna.058818.116. Epub 2017 Jan 9. RNA. 2017. PMID: 28069890 Free PMC article.
It has been long held that APOBEC1-mediated editing activity is dependent on the RNA binding protein A1CF. The function of A1CF in adult tissues has not been reported because a previously reported null allele displays embryonic lethality. ...
It has been long held that APOBEC1-mediated editing activity is dependent on the RNA binding protein A1CF. The function of A1CF in ad …
Genome-wide erasure of DNA methylation in mouse primordial germ cells is affected by AID deficiency.
Popp C, Dean W, Feng S, Cokus SJ, Andrews S, Pellegrini M, Jacobsen SE, Reik W. Popp C, et al. Nature. 2010 Feb 25;463(7284):1101-5. doi: 10.1038/nature08829. Nature. 2010. PMID: 20098412 Free PMC article.
The extent of this reprogramming and its molecular mechanisms are poorly understood. We previously showed that the cytidine deaminases AID and APOBEC1 can deaminate 5-methylcytosine in vitro and in Escherichia coli, and in the mouse are expressed in tissues in which …
The extent of this reprogramming and its molecular mechanisms are poorly understood. We previously showed that the cytidine deaminases AID a …
Activation-induced cytidine deaminase deaminates 5-methylcytosine in DNA and is expressed in pluripotent tissues: implications for epigenetic reprogramming.
Morgan HD, Dean W, Coker HA, Reik W, Petersen-Mahrt SK. Morgan HD, et al. J Biol Chem. 2004 Dec 10;279(50):52353-60. doi: 10.1074/jbc.M407695200. Epub 2004 Sep 24. J Biol Chem. 2004. PMID: 15448152 Free article.
The epigenetic modification by methylation of cytosine in CpG dinucleotides is also mutagenic, but this is thought to occur by spontaneous deamination. Here we show that Aid and Apobec1 are 5-methylcytosine deaminases resulting in a thymine base opposite a guanine. Their a …
The epigenetic modification by methylation of cytosine in CpG dinucleotides is also mutagenic, but this is thought to occur by spontaneous d …
An alternatively spliced form of apobec-1 messenger RNA is overexpressed in human colon cancer.
Lee RM, Hirano K, Anant S, Baunoch D, Davidson NO. Lee RM, et al. Gastroenterology. 1998 Nov;115(5):1096-103. doi: 10.1016/s0016-5085(98)70080-0. Gastroenterology. 1998. PMID: 9797364
RESULTS: Apobec-1 mRNA was detectable in normal and colon cancer tissue, metastatic nodules, and certain colon cancer-derived cell lines. ...CONCLUSIONS: Apobec-T is expressed in normal, adenomatous, and cancerous gastrointestinal tissues, and levels of the mRNA enc …
RESULTS: Apobec-1 mRNA was detectable in normal and colon cancer tissue, metastatic nodules, and certain colon cancer-derived cell li …
Apolipoprotein B RNA editing enzyme-deficient mice are viable despite alterations in lipoprotein metabolism.
Morrison JR, Pászty C, Stevens ME, Hughes SD, Forte T, Scott J, Rubin EM. Morrison JR, et al. Proc Natl Acad Sci U S A. 1996 Jul 9;93(14):7154-9. doi: 10.1073/pnas.93.14.7154. Proc Natl Acad Sci U S A. 1996. PMID: 8692961 Free PMC article.
The recent observations of tumor formation in Apobec-1 transgenic animals, together with the fact that Apobec-1 is expressed in numerous tissues lacking apo-B, raises the issue of whether this enzyme is essential for a variety of posttranscriptional editing events. To dire …
The recent observations of tumor formation in Apobec-1 transgenic animals, together with the fact that Apobec-1 is expressed in numerous …